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NDT Advance Access originally published online on January 26, 2008
Nephrology Dialysis Transplantation 2008 23(4):1101-1102; doi:10.1093/ndt/gfm933
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© The Author [2008]. Published by Oxford University Press on behalf of ERA-EDTA. All rights reserved. For Permissions, please e-mail: journals.permissions@oxfordjournals.org


Proximal RTA: Are all the charts completed yet?

Peter Gross and Constanze Meye

Division of Nephrology, Department of Medicine III, University Medical Center C. G. Carus, Dresden, Germany

Professor Peter Gross, M.D., Division of Nephrology, Department of Medicine III, Universitats Klinikum C. G. Carus Fetscherstrasse 74 01307 Dresden, Germany, Fax: 0049 351 458 5333, Tel: 0049 351 458 2645, E-mail: Peter.Gross@uniklinikum-dresden.de

Keywords: metabolic acidosis; renal tubular acidosis; tubular transport proteins

The first 10% of the full text of this article appears below.

Renal tubular acidosis (RTA) is an uncommon disorder; however, the subgroup of isolated familial proximal RTA (pRTA) is exceedingly rare. The term ‘isolated’ pRTA distinguishes these disorders from Fanconi syndrome, in which in addition to acid excretion, other proximal tubular functions are impaired as well. In this issue of Nephrology Dialysis Transplantation, Katzir et al. [1] report findings in a single family with a specific form of isolated familial pRTA. It is highly likely that only very few physicians will ever encounter a case of this nature in their professional lives. Why then would this report be of interest?

The human body generates ~50–100 mmol of . . . [Full Text of this Article]


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